Document Type

Article

Publication Date

10-5-2017

Comments

This article has been peer reviewed. It is the author’s final published version in Cell Death & Disease, Volume 8, Issue 10, October 2017, Page e3088.

The published version is available at https://doi.org/10.1038/cddis.2017.467. Copyright © Zhang et al.

Abstract

Oxidative stress impairs follicular development by inducing granulosa cell (GC) apoptosis, which involves enhancement of the transcriptional activity of the pro-apoptotic factor Forkhead box O1 (FoxO1). However, the mechanism by which oxidative stress promotes FoxO1 activity is still unclear. Here, we found that miR-181a was upregulated in hydrogen peroxide (H

Creative Commons License

Creative Commons License
This work is licensed under a Creative Commons Attribution 4.0 License.

Language

English

Available for download on Friday, October 05, 2018

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