Document Type

Article

Publication Date

5-11-2018

Comments

This article has been peer reviewed. It is the author’s final published version in Frontiers in Physiology, Volume 9, Issue MAY, May 2018, Article number 516.

The published version is available at https://doi.org/10.3389/fphys.2018.00516. Copyright © Zeng et al.

Abstract

Under inflammatory conditions, inflammatory cells release reactive oxygen species (ROS) and reactive nitrogen species (RNS) which cause DNA damage. If not appropriately repaired, DNA damage leads to gene mutations and genomic instability. DNA damage checkpoint factors (DDCF) and DNA damage repair factors (DDRF) play a vital role in maintaining genomic integrity. However, how DDCFs and DDRFs are modulated under physiological and pathological conditions are not fully known. We took an experimental database analysis to determine the expression of 26 DNA DD

Creative Commons License

Creative Commons License
This work is licensed under a Creative Commons Attribution 4.0 License.

Language

English

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